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There are many unanswered questions regarding both the functions of sleep and the effects of sleep loss. Sleep is considered to be important to body restitution, like energy conservation, thermoregulation, and tissue recovery (Maquet 2001). In addition, sleep is essential for cognitive performance, especially memory consolidation (Maquet 2001; Stickgold 2005). Sleep loss, instead, seems to activate the sympathetic nervous system, which can lead to a rise of blood pressure (Ogawa et al 2003) and an increase in cortisol secretion (Spiegel et al 1999; Lac and Chamoux 2003). Immune response may be impaired and metabolic changes such as insulin resistance may occur (for review, see Spiegel et al 2005). People who are exposed to sleep loss usually experience a decline in cognitive performance and changes in mood (for meta-analyses, see Pilcher and Huffcutt 1996; Philibert 2005).
In addition to the cognitive domains already introduced, total SD affects several other cognitive processes as well. It increases rigid thinking, perseveration errors, and difficulties in utilizing new information in complex tasks requiring innovative decision-making (Harrison and Horne 1999). Deterioration in decision-making also appears as more variable performance and applied strategies (Linde et al 1999), as well as more risky behavior (Killgore et al 2006). Several other tasks have been used in the sleep deprivation studies (Table 1). For example, motor function, rhythm, receptive and expressive speech, and memory measured with the Luria-Nebraska Neuropsychological Battery deteriorated after one night of SD, whereas tactile function, reading, writing, arithmetic and intellectual processes remain intact (Kim et al 2001).
Harrison and Horne (1998, 1999) suggest that the deterioration of cognitive performance during SD could be due to boredom and lack of motivation caused by repeated tasks, especially if the tests are simple and monotonous. They used short, novel, and interesting tasks to abolish this motivational gap, yet still noted that SD impaired performance. In contrast, other researchers suggest that sleep-deprived subjects could maintain performance in short tasks by being able to temporarily increase their attentional effort. When a task is longer, performance deteriorates as a function of time. A meta-analysis by Pilcher and Huffcutt (1996) provides support for that: total SD of less than 45 h deteriorated performance more severely in complex tasks with a long duration than in simple and short tasks. Based on this, it is probably necessary to make a distinction between mere attentional effort and more general motivation. Although attentional effort reflects motivational aspects in performance, motivation in a broader sense can be considered a long-term process such as achieving a previously set goal, eg, completing a study protocol. If one has already invested a great deal of time and effort in the participation, motivation to follow through may be increased.
It is difficult to compare the effects of total and partial SD based on existing literature due to large variation in methodologies, including the length of SD or the type of cognitive measures. The only study that has compared total and partial SD found that after controlling learning effects, cognitive performance declined almost linearly in the course of the study in all four experimental groups (Van Dongen et al 2003a): one group was exposed to 3 nights total SD, and in other experimental groups, time in bed was restricted to 4 or 6 h for 14 consecutive days. The control group was allowed 8 h in bed for 14 days. Impairment in psychomotor vigilance test and digit symbol substitution task for the 4 h group after 14 days was equal to that of the total SD group after 2 nights. Deterioration in the serial addition/subtraction task for the 4 h group was similar to that of the total SD group after 1 night. The effect of 6 h restricted sleep corresponded to 1 night of total SD in psychomotor vigilance and digit symbol. Performance remained unaffected in the control group.
Even though there is some evidence that older subjects tolerate SD better than young subjects, it is difficult to determine the age effect during SD with precision. However, because of age-related changes in many aspects of sleep and wakefulness, it is plausible that aging influences reactions to SD. As suggested previously, the weaker SD effect in aging may be due to attenuation of the circadian amplitude, which is reflected in the performance curve in vigilance tasks (Blatter et al 2006). Also, changes in the homeostatic process may play a role. During wakefulness, the accumulation of sleep pressure seems to be reduced in aging (Murillo-Rodriguez et al 2004), which could leave older subjects more alert. There is also evidence that aging subjects recover faster from SD than young subjects in terms of physiological sleep (Bonnet and Rosa 1987; Brendel et al 1990). This faster recovery in sleep state may also mean better restoration of cognitive performance (Bonnet and Rosa 1987; Brendel et al 1990). However, more research is necessary to confirm these hypotheses.
It is possible that physiological responses to SD are not equal among men and women. During SD of 38 h, EEG showed more sleep activity in men than in women during waking rest and cognitive performance (Corsi-Cabrera et al 2003). Presumably, therefore, one recovery night of nine hours would be enough to restore waking EEG activity in men, but not in women. Only a few studies have examined gender differences in cognitive performance during SD. In a vigilance task, performance was more impaired in men but returned to the baseline level in both men and women after recovery sleep (Corsi-Cabrera et al 2003). In another study, women performed better than men in verbal and in visuo-constructive tasks during 35 h SD (Binks et al 1999). No gender differences were observed in word fluency, maintenance or suppression of attention, auditory attention or cognitive flexibility. In that study, however, only one point of measurement was included, and so the difference in performance could be caused by SD or initial distinctions between the gender groups.
Few attempts have been made to evaluate the effect of sex hormones on coping with SD. It has been suggested that hormone therapy, which is widely used for women during their menopausal transition to help alleviate climacteric symptoms, attenuates physiological stress response (Lindheim et al 1992). However, after 25 h of total SD, no difference was observed between hormone therapy users and nonusers in visual episodic memory, visuomotor performance, verbal attention and shared attention (Alhola et al 2005). In addition, during 40 h of SD, hormone therapy did not produce any advantage in reaction time or vigilance tasks (Karakorpi et al 2006).
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